Researchers have identified and mapped diverse cell types in the cochlear nucleus, a critical part of the auditory system, advancing understanding of how our brains process sounds and paving the way for targeted treatments for auditory disorders.
The findings illustrate that discrete subcellular structures in the human organ of hearing, the cochlea, are involved in the variation of risk of age-related hearing loss in the population.
The cochlear duct houses the hearing organ, with many hair cells that are divided into outer and inner hair cells. The outer hair cells amplify sound vibrations, which enables us to hear faint sounds and perceive the various frequencies in human speech better. The inner hair cells convert the sound vibrations into nerve signals. In the current study, the researchers have investigated how the conversion takes place.
The data show hearing improvements in adults with acquired sensorineural hearing loss (SNHL) and the “first known linkage of pharmacokinetics and pharmacodynamics for a potential hearing restoration therapy,” according to Frequency.
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We hear sounds in part because tiny filaments inside our inner ears help convert voices, music, and noises into electrical signals that are sent to our brains for processing. Now, scientists have mapped and simulated those filaments at the atomic level, a discovery that shed lights on how the inner ear works and that could help researchers learn more about how and why people lose the ability to hear.
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At the end of two weeks, results showed that for the COVID-infected group, high-frequency pure-tone thresholds as well as the TEOAE amplitudes were significantly worse in the test group, indicating damage to outer ear hair cells.
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Vertidiag is said to have developed a technical platform to induce controlled vestibular disorders and to measure their functional impacts. This platform uses behavioral pharmacology, along with a battery of cellular and molecular biology tools that support the understanding of the mechanisms of action of potential drug candidates.
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FX-322 is Frequency’s lead product candidate, designed to regenerate auditory sensory hair cells in the cochlea and improve hearing in patients with sensorineural hearing loss (SNHL).
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Cochlear Limited (ASX: COH), a company that specializes in implantable hearing solutions,...
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Todays more competitive hearing care market makes it especially critical for practices to establish strong relationships with local physicians and heathcare providers.
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Cochlear Limited (ASX: COH), an implantable hearing solutions company, announced the US Food and...
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Using zebrafish as a proxy, scientists have shed light on how changes to specific genes alter the coordinated direction that these cells are laid out.
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In a Phase 1/2 study, FX-322 demonstrated a statistically significant and clinically meaningful improvement in key measures of hearing loss, including clarity of sound and word recognition, with no serious adverse events observed.
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Nearly 24% of US adults ages 20 to 69 have features of their hearing tests in one or both ears that suggest hearing loss from loud noise, based on a 2017 study by researchers from the NIDCD and the Centers for Disease Control and Prevention (CDC).
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The new technology is based on a recent hearing aid specification, Audio Streaming for Hearing Aids (ASHA) on Bluetooth Low Energy Connection-Oriented Channels, which Google has developed in collaboration with GN Hearing and Cochlear.
Read MoreThese funds will enable Strekin to conclude its ongoing Phase 3 clinical trial of STR001 in Sudden Sensorineural Hearing Loss, the RESTORE trial, and to prepare for a European filing.
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The proteins, described in a report published June 12 in “eLife,” may hold a key to future therapies to restore hearing in people with irreversible deafness. An article summarizing the research was published on the Johns Hopkins Medicine website.
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The researchers found that noise trauma causes substantially greater changes in neural processing of complex sounds compared with age-related metabolic loss, potentially explaining large differences in speech perception commonly seen between people with the same clinically defined degree of hearing loss based on an audiogram.
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